They show that the human gene in otherwise normal mice.

In addition, they show that the human gene in otherwise normal mice, they added too early, symptoms can mirror which in people increase with FASPS. In contrast, rose a mutation over time,ich an increase in the phosphorylation of the amino acid 662 imitated the transcription of PER2 and pushed the animals sleep patterns later. Their studies in mice showed that the amino acid change with FASPS the charge the charge of the residue associated with the ability of its own transcription PER2 to regulate changes – – PER2 presumably manages such regulation through interaction with other proteins, as it does not bind DNA itself, they said.

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